Please use this identifier to cite or link to this item:
https://repository.unad.edu.co/handle/10596/33248
Title: | Staphylococcus aureus: generalities, mechanisms of pathogenicity and cell colonization Staphylococcus aureus: generalidades, mecanismos de patogenicidad y colonización celular |
metadata.dc.creator: | Pasachova Garzón, Jennifer Ramirez Martinez , Sara Muñoz Molina , L |
Keywords: | Staphylococcus aureus;bacterial resistance;biofilm;MSCRAMM;fibronectin binding proteins;Staphylococcus aureus;resistencia bacteriana;biopelícula;MSCRAMM;proteínas de unión a la fibronectina |
Publisher: | Universidad Colegio Mayor de Cundinamarca |
metadata.dc.relation: | https://hemeroteca.unad.edu.co/index.php/nova/article/view/3631/3675 |
metadata.dc.format.*: | application/pdf |
metadata.dc.type: | info:eu-repo/semantics/article info:eu-repo/semantics/publishedVersion reviewArticle reviewArticle reviewArticle |
Description: | Staphylococcus aureus is a microorganism characterized by being the main cause of nosocomial bacteremia in different places of the world, due to the different virulence and pathogenicity factors. One of the most important is the biofilm formation, which greatly favors bacterial resistance. For the adhesion of the biofilm to biotic and abiotic surfaces, the microbial surface components recognizing adhesive matrix molecules (MSCRAMM), these proteins play a key role in host cell colonization and invasion by the bacteria. Staphylococcus aureus se caracteriza por ser la principal causa de bacteriemia nosocomial en el mundo, debido al incremento en la resistencia, a los diferentes factores de patogenicidad y virulencia y la expresión de una gran variedad de roteínas las cuales pertenecen a las moléculas de la matriz adhesiva (MSCRAMM), presentes en la superficie de la bacteria cuya función es la colonización e invasión celular al hospedero y favorecer la formación de biopelícula, El conjunto de estos mecanismos de patogenicidad y virulencia, le permiten a la bacteria persistir en el huésped y en el ambiente, sobreviviendo a factores adversos, al sistema inmune y a los antimicrobianos. |
metadata.dc.source: | NOVA Biomedical Sciences Journal; Vol. 17 No. 32 (2019); 25 - 38 Nova; Vol. 17 Núm. 32 (2019); 25 - 38 NOVA Ciências Biomédicas Publicação; v. 17 n. 32 (2019); 25 - 38 2462-9448 1794-2470 |
Other Identifiers: | https://hemeroteca.unad.edu.co/index.php/nova/article/view/3631 10.22490/24629448.3631 |
Appears in Collections: | Revista Nova |
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